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On one hand, FXR protects the liver from bile acid overload by repressing bile salt synthesis through the inhibition of CYP7A1, a process mediated by the induction of small heterodimer partner (SHP)

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In this review, we will examine the phenomenon of receptor dimerization with a focus on the role of the ghrelin receptor in such dimers and how this knowledge can aid in devising improved treatments for such disorders as Parkinsons disease, schizophrenia, obesity, depression, and diabetes